Neuroscience News

Eight Stanford University scientists, including SNI Affiliates Michael Lin, Thomas Rando, and Tony Wyss-Coray, have received more than $17 million from the National Institutes of Health that will enable them to pursue innovative research in biomedicine.

Scientists have shown how a protein fragment known as beta-amyloid, strongly implicated in Alzheimer’s disease, begins destroying synapses before it clumps into plaques that lead to nerve cell death.

Researchers have shown that oxytocin — often referred to as "the love hormone" because of its importance in the formation and maintenance of strong mother-child and sexual attachments — is involved in a broader range of social interactions than previously

A defective trash-disposal system in the brain’s resident immune cells may be a major contributor to neurodegenerative disease. Preliminary observations show that this defect appears in the brains of patients who died of Alzheimer’s disease, so correcting

The steady accumulation of a protein in healthy, aging brains may explain seniors’ vulnerability to neurodegenerative disorders, a new study by researchers at the Stanford University School of Medicine reports.

Long-term hearing loss from loud explosions, such as blasts from roadside bombs, may not be as irreversible as previously thought, according to a new study by researchers at the Stanford University School of Medicine.

A chemical code scrawled on histones — the protein husks that coat DNA in every animal or plant cell — determines which genes in that cell are turned on and which are turned off. Now, Stanford researchers have taken a new step in the deciphering of that h

Researchers at the Stanford University School of Medicine have found that a naturally occurring protein secreted only in discrete areas of the mammalian brain may act as a Valium-like brake on certain types of epileptic seizures.

Stanford scientists have created a mouse model of muscular dystrophy in which degenerating muscle tissue gives off visible light.

Healthy menopausal women carrying a well-known genetic risk factor for Alzheimer’s disease showed measurable signs of accelerated biological aging, a new study has found.